導讀:
反式脂肪,又稱為反式脂肪酸、逆態脂肪酸
(Trans fat, also called trans-unsaturated fatty acids or trans fatty acids)
,屬於不飽和脂肪酸(單元不飽和或多元不飽和)。
反式脂肪酸屬於不飽和脂肪酸,曾被認為是較飽和脂肪酸更健康,
反式脂肪將會提高罹患冠狀動脈心臟病的機率,因為它可令低密度脂蛋白上升,
並使高密度脂蛋白下降。
trans fatty acids may lower high-density lipoprotein (HDL) cholesterol
levels and raise triglyceride and lipoprotein(a) levels in plasma.
反式脂肪酸可降低高密度脂蛋白(HDL)膽固醇水平並提高血漿中的甘油三酯
和脂蛋白(低密度)水平。
Among persons whose diets were high in saturated and trans fats,
higher copper intake was associated with a faster rate of
cognitive decline.
在飲食中飽和脂肪和反式脂肪含量高的人群中,銅攝入量越高,
認知能力下降速度越快。
所以銅雖對人體有正面作用,但對反式脂肪沒有正面幫助。
Engineered Lipase Cuts Out Trans Fats
工程脂肪酶切斷反式脂肪
因此人體的脂肪酶應該也能分解反式脂肪
Our model indicated that a diet inadequate in magnesium combined
with trans fat may increase the risk of calcification of endothelial
cells.
我們的模型表明,鎂不足和反式脂肪的飲食可能會增加內皮細胞鈣化的風險。
Magnesium is necessary the activity of lecithin cholesterol acyltransferase
(LCAT) and lipoprotein lipase (LPL), which lowers triglyceride levels
and raises HDL-cholesterol levels.
鎂是卵磷脂膽固醇酰基轉移酶(LCAT)和脂蛋白脂肪酶(LPL)的活性所必需的,
其降低甘油三酯水平並提高HDL(高密度脂蛋白)-膽固醇水平。
post-heparin lipase activity (PHLA)
後肝素脂肪酶活性(PHLA)
Mg-deficient and control diets
缺鎂和對照飲食
PHLA in deficient rat was substantially lower than in control rats.
缺陷(缺鎂)大鼠的PHLA顯著低於對照大鼠。
The inverse correlation between plasma triglyceride concentration
and PHLA strongly suggests that hypertriglyceridemia
is the result of defective lipolysis of plasma triglycerides
in Mg-deficient rats.
血漿甘油三酯濃度與PHLA之間的負相關強烈表明高甘油三酯血症是缺鎂
大鼠血漿甘油三酯缺乏脂解的結果。
Lipases LipA and LipB
脂肪酶LipA和LipB
Metal analysis indicated that both LipA and LipB contain 1 Ca2+
and one Mn2+ ion per monomeric enzyme unit.
金屬分析表明LipA和LipB每個單體酶單元含有1個Ca2+(鈣)和1個Mn2+(錳)離子。
Salts of manganese, magnesium, sodium and calcium stimulated lipase activity.
錳,鎂,和鈣的鹽刺激脂肪酶活性
總結以上:
脂肪酶應能協助分解反式脂肪
而脂肪酶活性應該主要和鎂、錳、鈣相關
建議可以喝紅茶(含鎂、錳)