纖維蛋白溶酶原激活物是一種絲氨酸蛋白酶,它可以將纖溶蛋白酶原轉化為纖溶蛋白酶,從而促進纖維蛋白溶解。
Inhibition patterns indicated that the enzyme was a serine protease with no
metal dependence, although it was stabilized by magnesium ions.
抑制模式表明該酶是絲氨酸蛋白酶,沒有金屬依賴性,儘管它被鎂離子穩定。
Fluctuations in the fibrinolytic parameters were maintained in the magnesium group
纖維蛋白溶解參數的波動在鎂組中維持
Mg deficiency is a stimulative factor of hepatic fibrosis and steatosis possibly
via its effect on mast cells.
缺鎂是肝纖維化和脂肪變性的促進因素,可能是通過其對肥大細胞的影響。
In a group of 50 patients with liver cirrhosis compared with a group of
50 clinically healthy subjects serum magnesium levels were determined.
在50名肝硬化患者組中,與50名臨床健康受試者組相比,測定血清鎂水平。
The patients present a significant decrease of serum magnesium levels in comparison to controls.
與對照相比,患者血清鎂水平顯著降低。
The patients with alcoholic cirrhosis of the liver and with ascite have significant lower magnesium levels
患有酒精性肝硬化和腹水的患者的鎂水平顯著降低。
二、
Rat hepatic fibrosis was developed successfully. The fibrosis changes were partially
reversed by simultaneous administration of IL-10.
大鼠肝纖維化成功發展。 通過同時施用IL-10部分逆轉纖維化變化。
Effect of interferon-gamma on hepatic fibrosis in chronic hepatitis B virus infection
干擾素-γ對慢性乙型肝炎病毒感染肝纖維化的影響
Fifty-four patients in the IFN-gamma group and 29 patients in the control group completed the study.
IFN-γ組中的54名患者和對照組中的29名患者完成了該研究。
The hepatic fibrosis score was significantly reduced in 63% of IFN-gamma treated
patients compared with 24.1% in the control group
63%的IFN-γ治療患者的肝纖維化評分顯著降低,而對照組為24.1%。
能提高IL-10的元素:鋅、錳、硒、維生素A、維生素D、維生素E
能提高IFN-γ的元素:鋅、硒、錳、維生素C、高劑量維生素E
與導讀吻合的有鋅、硒、維生素D
總結以上:
綜合一、二,肝硬化可逆轉,缺鎂鋅硒維它命D,建議一天一粒善存+多喝豆漿。
含鎂高的食物(由多至少依序排列):海苔、松子、榛果、亞麻籽、南瓜子、甘草、小茴香、桑葚